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Primary murine Chlamydia trachomatis pneumonia in B-cell-deficient mice.

Mice were rendered deficient in B-cell activity by treatment with anti-mu antibody from birth. These animals were then infected intranasally with murine Chlamydia trachomatis (murine pneumonitis agent [MoPn]). They produced neither local nor systemic antibody to MoPn but had intact delayed-type hype...

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Dades bibliogràfiques
Publicat a:Infect Immun
Autors principals: Williams, D M, Grubbs, B, Schachter, J
Format: Artigo
Idioma:Inglês
Publicat: American Society for Microbiology (ASM) 1987
Matèries:
Accés en línia:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC260718/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/3498690/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.55.10.2387-2390.1987
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