Primary murine Chlamydia trachomatis pneumonia in B-cell-deficient mice.

Mice were rendered deficient in B-cell activity by treatment with anti-mu antibody from birth. These animals were then infected intranasally with murine Chlamydia trachomatis (murine pneumonitis agent [MoPn]). They produced neither local nor systemic antibody to MoPn but had intact delayed-type hype...

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Pubblicato in:	Infect Immun
Autori principali:	Williams, D M, Grubbs, B, Schachter, J
Natura:	Artigo
Lingua:	Inglês
Pubblicazione:	American Society for Microbiology (ASM) 1987
Soggetti:	Research Article
Accesso online:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC260718/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/3498690/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.55.10.2387-2390.1987
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