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Primary murine Chlamydia trachomatis pneumonia in B-cell-deficient mice.

Mice were rendered deficient in B-cell activity by treatment with anti-mu antibody from birth. These animals were then infected intranasally with murine Chlamydia trachomatis (murine pneumonitis agent [MoPn]). They produced neither local nor systemic antibody to MoPn but had intact delayed-type hype...

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Bibliographic Details
Main Authors: Williams, D M, Grubbs, B, Schachter, J
Format: Artigo
Language:Inglês
Published: 1987
Subjects:
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC260718/
https://ncbi.nlm.nih.gov/pubmed/3498690
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