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c-Abl is required for development and optimal cell proliferation in the context of p53 deficiency

The c-Abl tyrosine kinase and the p53 tumor suppressor protein interact functionally and biochemically in cellular genotoxic stress response pathways and are implicated as downstream mediators of ATM (ataxia-telangiectasia mutated). This fact led us to study genetic interactions in vivo between c-Ab...

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Detaylı Bibliyografya
Asıl Yazarlar: Whang, Young E., Tran, Chris, Henderson, Cailin, Syljuasen, Randi G., Rozengurt, Nora, McBride, William H., Sawyers, Charles L.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: The National Academy of Sciences 2000
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC25855/
https://ncbi.nlm.nih.gov/pubmed/10805805
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