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Defective homing is associated with altered Cdc42 activity in cells from patients with Fanconi anemia group A
Previous studies showed that Fanconi anemia (FA) murine stem cells have defective reconstitution after bone marrow (BM) transplantation. The mechanism underlying this defect is not known. Here, we report defective homing of FA patient BM progenitors transplanted into mouse models. Using cells from p...
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| Asıl Yazarlar: | , , , , , , , , , , |
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| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
American Society of Hematology
2008
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2518878/ https://ncbi.nlm.nih.gov/pubmed/18565850 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2008-03-147090 |
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