The death domain-containing kinase RIP1 regulates p27(Kip1) levels through the PI3K–Akt–forkhead pathway

Elucidating the cross-talk between inflammatory and cell proliferation pathways might provide important insights into the pathogenesis of inflammation-induced cancer. Here, we show that the receptor-interacting protein 1 (RIP1)—an essential mediator of inflammation-induced nuclear factor-κB (NF-κB)...

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Bibliographische Detailangaben
Hauptverfasser: Park, Seongmi, Ramnarain, Deepti B, Hatanpaa, Kimmo J, Mickey, Bruce E, Saha, Debabrata, Paulmurugan, Ramasamy, Madden, Christopher J, Wright, Paul S, Bhai, Salman, Ali, M Aktar, Puttaparthi, Krishna, Hu, Wei, Elliott, Jeffrey L, Stuve, Olaf, Habib, Amyn A
Format: Artigo
Sprache:Inglês
Veröffentlicht: Nature Publishing Group 2008
Schlagworte:
Scientific Report
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2515213/
https://ncbi.nlm.nih.gov/pubmed/18566599
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/embor.2008.102
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