Properties of a simian virus 40 mutant T antigen substituted in the hydrophobic region: defective ATPase and oligomerization activities and altered phosphorylation accompany an inability to complex with cellular p53.

Lignende værker

• A DNA replication-positive mutant of simian virus 40 that is defective for transformation and the production of infectious virions.
  af: Peden, K W, et al.
  Udgivet: (1990)
• The large tumor antigen of simian virus 40 encodes at least two distinct transforming functions.
  af: Srinivasan, A, et al.
  Udgivet: (1989)
• Site-directed mutagenesis of the simian virus 40 large T-antigen gene: replication-defective amino acid substitution mutants that retain the ability to induce morphological transformation.
  af: Peden, K W, et al.
  Udgivet: (1985)
• Simian virus 40 T antigen can regulate p53-mediated transcription independent of binding p53.
  af: Rushton, J J, et al.
  Udgivet: (1997)
• Phosphorylation of p53 in normal and simian virus 40-transformed NIH 3T3 cells.
  af: Meek, D W, et al.
  Udgivet: (1988)