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IGF-1 regulates cAMP levels in astrocytes through a β(2)-adrenergic receptor-dependant mechanism

We have recently demonstrated that neonatal astrocytes derived from mice lacking beta-2 adrenergic receptors (β(2)AR) possess higher proliferation rates, as compared to wild-type cells, an attribute that was shown to involve insulin-like growth factor (IGF) signaling. In the present study, we demons...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: Chesik, Daniel, Wilczak, Nadine, De Keyser, Jacques
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Ivyspring International Publisher 2008
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2500150/
https://ncbi.nlm.nih.gov/pubmed/18690292
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