RAGE Mediates Podocyte Injury in Adriamycin-induced Glomerulosclerosis

In the kidney, the receptor for advanced glycation end products (RAGE) is principally expressed in the podocyte at low levels, but is upregulated in both human and mouse glomerular diseases. Because podocyte injury is central to proteinuric states, such as the nephrotic syndrome, the murine adriamyc...

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Bibliographische Detailangaben
Hauptverfasser: Guo, Jiancheng, Ananthakrishnan, Radha, Qu, Wu, Lu, Yan, Reiniger, Nina, Zeng, Shan, Ma, Wanchao, Rosario, Rosa, Yan, Shi Fang, Ramasamy, Ravichandran, D'Agati, Vivette, Marie Schmidt, Ann
Format: Artigo
Sprache:Inglês
Veröffentlicht: American Society of Nephrology 2008
Schlagworte:
Basic Research
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2386730/
https://ncbi.nlm.nih.gov/pubmed/18256352
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2007101109
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