RAGE Mediates Podocyte Injury in Adriamycin-induced Glomerulosclerosis

In the kidney, the receptor for advanced glycation end products (RAGE) is principally expressed in the podocyte at low levels, but is upregulated in both human and mouse glomerular diseases. Because podocyte injury is central to proteinuric states, such as the nephrotic syndrome, the murine adriamyc...

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Detalhes bibliográficos
Main Authors: Guo, Jiancheng, Ananthakrishnan, Radha, Qu, Wu, Lu, Yan, Reiniger, Nina, Zeng, Shan, Ma, Wanchao, Rosario, Rosa, Yan, Shi Fang, Ramasamy, Ravichandran, D'Agati, Vivette, Marie Schmidt, Ann
Formato: Artigo
Idioma:Inglês
Publicado em: American Society of Nephrology 2008
Assuntos:
Basic Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2386730/
https://ncbi.nlm.nih.gov/pubmed/18256352
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2007101109
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