Targeting homeostatic mechanisms of endoplasmic reticulum stress to increase susceptibility of cancer cells to fenretinide-induced apoptosis: the role of stress proteins ERdj5 and ERp57

Endoplasmic reticulum (ER) malfunction, leading to ER stress, can be a consequence of genome instability and hypoxic tissue environments. Cancer cells survive by acquiring or enhancing survival mechanisms to counter the effects of ER stress and these homeostatic responses may be new therapeutic targ...

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Detalhes bibliográficos
Main Authors: Corazzari, M, Lovat, P E, Armstrong, J L, Fimia, G M, Hill, D S, Birch-Machin, M, Redfern, C P F, Piacentini, M
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group 2007
Assuntos:
Translational Therapeutics
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2360126/
https://ncbi.nlm.nih.gov/pubmed/17353921
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.bjc.6603672
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