Caricamento...
Molecular Mechanism for ATP-Dependent Closure of the K(+) Channel Kir6.2
In the ATP-dependent K(+) (K(ATP)) channel pore-forming protein Kir6.2, mutation of three positively charged residues, R50, K185 and R201, impairs the ability of ATP to close the channel. The mutations do not change the channel open probability (P(o)) in the absence of ATP, supporting the involvemen...
Salvato in:
| Autori principali: | , , , |
|---|---|
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Blackwell Science Inc
2003
|
| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2343328/ https://ncbi.nlm.nih.gov/pubmed/12860923 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2003.048843 |
| Tags: |
Aggiungi Tag
Nessun Tag, puoi essere il primo ad aggiungerne! !
|