Function of the c-Myc antagonist Mad1 during a molecular switch from proliferation to differentiation.

Mad-Max heterodimers have been shown to antagonize Myc transforming activity by a mechanism requiring multiple protein-protein and protein-DNA interactions. However, the mechanism by which Mad functions in differentiation is unknown. Here, we present evidence that Mad functions by an active repressi...

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Bibliographische Detailangaben
Hauptverfasser: Cultraro, C M, Bino, T, Segal, S
Format: Artigo
Sprache:Inglês
Veröffentlicht: 1997
Schlagworte:
Research Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC232084/
https://ncbi.nlm.nih.gov/pubmed/9111304
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