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Targeted disruption of the MYC antagonist MAD1 inhibits cell cycle exit during granulocyte differentiation.
The switch from transcriptionally activating MYC-MAX to transcriptionally repressing MAD1-MAX protein heterodimers has been correlated with the initiation of terminal differentiation in many cell types. To investigate the function of MAD1-MAX dimers during differentiation, we disrupted the Mad1 gene...
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| Hoofdauteurs: | , , , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
1998
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1170426/ https://ncbi.nlm.nih.gov/pubmed/9451002 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/17.3.774 |
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