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A point mutation in the γ(2) subunit of γ-aminobutyric acid type A receptors results in altered benzodiazepine binding site specificity
Benzodiazepines allosterically modulate γ-aminobutyric acid (GABA) evoked chloride currents of γ-aminobutyric acid type A (GABA(A)) receptors. Coexpression of either rat γ(2) or γ(3), in combination with α(1) and β(2) subunits, results both in receptors displaying high [(3)H]Ro 15-1788 affinity. How...
Tallennettuna:
| Päätekijät: | , |
|---|---|
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
The National Academy of Sciences of the USA
1997
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC23149/ https://ncbi.nlm.nih.gov/pubmed/9238062 |
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