The extracellular signal-regulated kinase pathway phosphorylates AML1, an acute myeloid leukemia gene product, and potentially regulates its transactivation ability.

AML1 (also called PEBP2alphaB, CBFA2, or CBFalpha2) is one of the most frequently disrupted genes in chromosome abnormalities seen in human leukemias. It has been reported that AML1 plays several pivotal roles in myeloid hematopoietic differentiation and other biological phenomena, probably through...

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Detaylı Bibliyografya
Yayımlandı:	Mol Cell Biol
Asıl Yazarlar:	Tanaka, T, Kurokawa, M, Ueki, K, Tanaka, K, Imai, Y, Mitani, K, Okazaki, K, Sagata, N, Yazaki, Y, Shibata, Y, Kadowaki, T, Hirai, H
Materyal Türü:	Artigo
Dil:	Inglês
Baskı/Yayın Bilgisi:	Taylor & Francis 1996
Konular:	Research Article
Online Erişim:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC231393/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8668214/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.16.7.3967
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The extracellular signal-regulated kinase pathway phosphorylates AML1, an acute myeloid leukemia gene product, and potentially regulates its transactivation ability.

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