Host PrP Glycosylation: A Major Factor Determining the Outcome of Prion Infection

Lignende værker

• Glycosylation of PrP(C) Determines Timing of Neuroinvasion and Targeting in the Brain following Transmissible Spongiform Encephalopathy Infection by a Peripheral Route
  af: Cancellotti, Enrico, et al.
  Udgivet: (2010)
• Dramatic Reduction of PrP(C) Level and Glycosylation in Peripheral Nerves following PrP Knock-Out from Schwann Cells Does Not Prevent Transmissible Spongiform Encephalopathy Neuroinvasion
  af: Bradford, Barry M., et al.
  Udgivet: (2009)
• PrP aggregation can be seeded by pre-formed recombinant PrP amyloid fibrils without the replication of infectious prions
  af: Barron, Rona M., et al.
  Udgivet: (2016)
• Increased Susceptibility of Human-PrP Transgenic Mice to Bovine Spongiform Encephalopathy Infection following Passage in Sheep
  af: Plinston, Chris, et al.
  Udgivet: (2011)
• Prion Propagation in Cells Expressing PrP Glycosylation Mutants
  af: Salamat, Muhammad K., et al.
  Udgivet: (2011)