Protein–protein interactions between triadin and calsequestrin are involved in modulation of sarcoplasmic reticulum calcium release in cardiac myocytes

In cardiac muscle, intracellular Ca(2+) release is controlled by a number of proteins including the ryanodine receptor (RyR2), calsequestrin (CASQ2), triadin-1 (Trd) and junctin (Jn) which form a complex in the junctional sarcoplasmic reticulum (SR) membrane. Within this complex, Trd appears to link...

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Detalhes bibliográficos
Main Authors: Terentyev, Dmitry, Viatchenko-Karpinski, Serge, Vedamoorthyrao, Srikanth, Oduru, Sridhar, Györke, Inna, Williams, Simon C, Györke, Sandor
Formato: Artigo
Idioma:Inglês
Publicado em: Blackwell Science Inc 2007
Assuntos:
Cellular
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2277233/
https://ncbi.nlm.nih.gov/pubmed/17569730
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2007.136879
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