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Prion protein insertional mutations increase aggregation propensity but not fiber stability

BACKGROUND: Mutations in the PRNP gene account for ~15% of all prion disease cases. Little is understood about the mechanism of how some of these mutations in PRNP cause the protein to aggregate into amyloid fibers or cause disease. We have taken advantage of a chimeric protein system to study the o...

Ausführliche Beschreibung

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Bibliographische Detailangaben
Hauptverfasser: Kalastavadi, Tejas, True, Heather L
Format: Artigo
Sprache:Inglês
Veröffentlicht: BioMed Central 2008
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2276218/
https://ncbi.nlm.nih.gov/pubmed/18366654
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1471-2091-9-7
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