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Glucocorticoids differentially regulate degradation of MyoD and Id1 by N-terminal ubiquitination to promote muscle protein catabolism

Accelerated protein degradation via the ubiquitin–proteasome pathway is the principal cause of skeletal muscle wasting associated with common human disease states and pharmacological treatment with glucocorticoids. Although many protein regulatory factors essential for muscle development and regener...

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書目詳細資料
Main Authors: Sun, Liping, Trausch-Azar, Julie S., Muglia, Louis J., Schwartz, Alan L.
格式: Artigo
語言:Inglês
出版: National Academy of Sciences 2008
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在線閱讀:https://ncbi.nlm.nih.gov/pmc/articles/PMC2265166/
https://ncbi.nlm.nih.gov/pubmed/18296633
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0800165105
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