The Estrogen Effects on Endothelial Repair and Mitogen-Activated Protein Kinase Activation Are Abolished in Endothelial Nitric-Oxide (NO) Synthase Knockout Mice, but Not by NO Synthase Inhibition by N-Nitro-l-arginine Methyl Ester

We have previously shown that estrogen exerts a vasoprotective effect by accelerating reendothelialization after perivascular artery injury through activation of the estrogen receptor α. Because 17β-estradiol (E(2)) is known to increase the bioavailability of nitric oxide, in this study, we used the...

وصف كامل

محفوظ في:
التفاصيل البيبلوغرافية
المؤلفون الرئيسيون: Billon, Audrey, Lehoux, Stéphanie, Lam Shang Leen, Laetitia, Laurell, Henrik, Filipe, Cédric, Benouaich, Vincent, Brouchet, Laurent, Dessy, Chantal, Gourdy, Pierre, Gadeau, Alain-Pierre, Tedgui, Alain, Balligand, Jean-Luc, Arnal, Jean-François
التنسيق: Artigo
اللغة:Inglês
منشور في: American Society for Investigative Pathology 2008
الموضوعات:
Regular Articles
الوصول للمادة أونلاين:https://ncbi.nlm.nih.gov/pmc/articles/PMC2258270/
https://ncbi.nlm.nih.gov/pubmed/18276789
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2353/ajpath.2008.070439
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