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Hypercholesterolemia decreases nitric oxide production by promoting the interaction of caveolin and endothelial nitric oxide synthase

Hypercholesterolemia is a central pathogenic factor of endothelial dysfunction caused in part by an impairment of endothelial nitric oxide (NO) production through mechanisms that remain poorly characterized. The activity of the endothelial isoform of NO synthase (eNOS) was recently shown to be modul...

詳細記述

保存先:
書誌詳細
主要な著者: Feron, Olivier, Dessy, Chantal, Moniotte, Stephane, Desager, Jean-Pierre, Balligand, Jean-Luc
フォーマット: Artigo
言語:Inglês
出版事項: American Society for Clinical Investigation 1999
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC408139/
https://ncbi.nlm.nih.gov/pubmed/10079111
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