Hypercholesterolemia decreases nitric oxide production by promoting the interaction of caveolin and endothelial nitric oxide synthase

Hypercholesterolemia is a central pathogenic factor of endothelial dysfunction caused in part by an impairment of endothelial nitric oxide (NO) production through mechanisms that remain poorly characterized. The activity of the endothelial isoform of NO synthase (eNOS) was recently shown to be modul...

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Bibliografische gegevens

Gepubliceerd in:	J Clin Invest
Hoofdauteurs:	Feron, Olivier, Dessy, Chantal, Moniotte, Stephane, Desager, Jean-Pierre, Balligand, Jean-Luc
Formaat:	Artigo
Taal:	Inglês
Gepubliceerd in:	American Society for Clinical Investigation 1999
Onderwerpen:	Article
Online toegang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC408139/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10079111/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI4829
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