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Hypercholesterolemia decreases nitric oxide production by promoting the interaction of caveolin and endothelial nitric oxide synthase

Hypercholesterolemia is a central pathogenic factor of endothelial dysfunction caused in part by an impairment of endothelial nitric oxide (NO) production through mechanisms that remain poorly characterized. The activity of the endothelial isoform of NO synthase (eNOS) was recently shown to be modul...

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Autors principals: Feron, Olivier, Dessy, Chantal, Moniotte, Stephane, Desager, Jean-Pierre, Balligand, Jean-Luc
Format: Artigo
Idioma:Inglês
Publicat: American Society for Clinical Investigation 1999
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC408139/
https://ncbi.nlm.nih.gov/pubmed/10079111
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