Aβ42 Mutants with Different Aggregation Profiles Induce Distinct Pathologies in Drosophila

Lignende værker

• Overexpression of Neprilysin Reduces Alzheimer Amyloid-β42 (Aβ42)-induced Neuron Loss and Intraneuronal Aβ42 Deposits but Causes a Reduction in cAMP-responsive Element-binding Protein-mediated Transcription, Age-dependent Axon Pathology, and Premature Death in Drosophila
  af: Iijima-Ando, Kanae, et al.
  Udgivet: (2008)
• Distinctive roles of different β-amyloid 42 aggregates in modulation of synaptic functions
  af: Chiang, Hsueh-Cheng, et al.
  Udgivet: (2009)
• Mitochondrial Mislocalization Underlies Aβ42-Induced Neuronal Dysfunction in a Drosophila Model of Alzheimer's Disease
  af: Iijima-Ando, Kanae, et al.
  Udgivet: (2009)
• Tau Ser262 phosphorylation is critical for Aβ42-induced tau toxicity in a transgenic Drosophila model of Alzheimer's disease
  af: Iijima, Koichi, et al.
  Udgivet: (2010)
• Regulation of Energy Stores and Feeding by Neuronal and Peripheral CREB Activity in Drosophila
  af: Iijima, Koichi, et al.
  Udgivet: (2009)