Potentiation of N-Methyl-d-Aspartate receptor-mediated neuronal injury during methamphetamine withdrawal in vitro requires co-activation of IP(3) receptors

Antzeko izenburuak

• Inhibition of sigma 1 receptor reduces N-methyl-d-aspartate induced neuronal injury in methamphetamine-exposed and -naive hippocampi
  nork: Smith, Katherine J., et al.
  Argitaratua: (2010)
• Methamphetamine exposure antagonizes N-methyl-D-aspartate receptor-mediated neurotoxicity in organotypic hippocampal slice cultures
  nork: Smith, Katherine J., et al.
  Argitaratua: (2007)
• Sex Differences in the Neurotoxic Effects of Adenosine A(1) Receptor Antagonism During Ethanol Withdrawal: Reversal With an A(1) Receptor Agonist or an NMDA Receptor Antagonist
  nork: Butler, Tracy R., et al.
  Argitaratua: (2008)
• Intra-cornu ammonis 1 administration of the human immunodeficiency virus-1 transcription factor Tat exacerbates the ethanol withdrawal syndrome in rodents and activates N-methyl-d-aspartate glutamate receptors to produce persisting spatial learning deficits
  nork: Self, Rachel L., et al.
  Argitaratua: (2009)
• Ethanol withdrawal is required to produce persisting N-methyl-D-aspartate receptor-dependent hippocampal cytotoxicity during chronic intermittent ethanol exposure
  nork: Reynolds, Anna R., et al.
  Argitaratua: (2015)