טוען...
Potentiation of N-Methyl-d-Aspartate receptor-mediated neuronal injury during methamphetamine withdrawal in vitro requires co-activation of IP(3) receptors
Recent findings suggest that methamphetamine (METH) functions acutely to inhibit N-methyl-d-aspartate (NMDA) receptor function. Protracted withdrawal from METH exposure may increase the sensitivity of NMDA receptors to agonist exposure, promoting neuronal excitability. However, the relevance of METH...
שמור ב:
| Main Authors: | , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2007
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2184481/ https://ncbi.nlm.nih.gov/pubmed/18021755 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.brainres.2007.10.019 |
| תגים: |
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