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Potentiation of N-Methyl-d-Aspartate receptor-mediated neuronal injury during methamphetamine withdrawal in vitro requires co-activation of IP(3) receptors

Recent findings suggest that methamphetamine (METH) functions acutely to inhibit N-methyl-d-aspartate (NMDA) receptor function. Protracted withdrawal from METH exposure may increase the sensitivity of NMDA receptors to agonist exposure, promoting neuronal excitability. However, the relevance of METH...

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שמור ב:
מידע ביבליוגרפי
Main Authors: Smith, Katherine J., Butler, Tracy R., Self, Rachel L., Braden, Brittany B., Prendergast, Mark A.
פורמט: Artigo
שפה:Inglês
יצא לאור: 2007
נושאים:
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC2184481/
https://ncbi.nlm.nih.gov/pubmed/18021755
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.brainres.2007.10.019
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