Potentiation of N-Methyl-d-Aspartate receptor-mediated neuronal injury during methamphetamine withdrawal in vitro requires co-activation of IP(3) receptors

Samankaltaisia teoksia

• Inhibition of sigma 1 receptor reduces N-methyl-d-aspartate induced neuronal injury in methamphetamine-exposed and -naive hippocampi
  Tekijä: Smith, Katherine J., et al.
  Julkaistu: (2010)
• Methamphetamine exposure antagonizes N-methyl-D-aspartate receptor-mediated neurotoxicity in organotypic hippocampal slice cultures
  Tekijä: Smith, Katherine J., et al.
  Julkaistu: (2007)
• Sex Differences in the Neurotoxic Effects of Adenosine A(1) Receptor Antagonism During Ethanol Withdrawal: Reversal With an A(1) Receptor Agonist or an NMDA Receptor Antagonist
  Tekijä: Butler, Tracy R., et al.
  Julkaistu: (2008)
• Intra-cornu ammonis 1 administration of the human immunodeficiency virus-1 transcription factor Tat exacerbates the ethanol withdrawal syndrome in rodents and activates N-methyl-d-aspartate glutamate receptors to produce persisting spatial learning deficits
  Tekijä: Self, Rachel L., et al.
  Julkaistu: (2009)
• Ethanol withdrawal is required to produce persisting N-methyl-D-aspartate receptor-dependent hippocampal cytotoxicity during chronic intermittent ethanol exposure
  Tekijä: Reynolds, Anna R., et al.
  Julkaistu: (2015)