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IL-23 produced by CNS-resident cells controls T cell encephalitogenicity during the effector phase of experimental autoimmune encephalomyelitis

CNS-resident cells, in particular microglia and macrophages, are a source of inflammatory cytokines during inflammation within the CNS. Expression of IL-23, a recently discovered cytokine, has been shown to be critical for the development of experimental autoimmune encephalomyelitis (EAE) in mice. E...

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Bibliografski detalji
Glavni autori: Becher, Burkhard, Durell, Brigit G., Noelle, Randolph J.
Format: Artigo
Jezik:Inglês
Izdano: American Society for Clinical Investigation 2003
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC213492/
https://ncbi.nlm.nih.gov/pubmed/14561703
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI200319079
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