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IL-23 produced by CNS-resident cells controls T cell encephalitogenicity during the effector phase of experimental autoimmune encephalomyelitis
CNS-resident cells, in particular microglia and macrophages, are a source of inflammatory cytokines during inflammation within the CNS. Expression of IL-23, a recently discovered cytokine, has been shown to be critical for the development of experimental autoimmune encephalomyelitis (EAE) in mice. E...
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| Main Authors: | , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Clinical Investigation
2003
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| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC213492/ https://ncbi.nlm.nih.gov/pubmed/14561703 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI200319079 |
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