Aberrant infection and persistence of varicella-zoster virus in human dorsal root ganglia in vivo in the absence of glycoprotein I

Varicella-zoster virus (VZV) causes varicella, establishes latency in sensory ganglia, and reactivates as herpes zoster. Human dorsal root ganglia (DRGs) xenografts in immunodeficient mice provide a model for evaluating VZV neuropathogenesis. Our investigation of the role of glycoprotein I (gI), whi...

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Detaylı Bibliyografya
Asıl Yazarlar: Zerboni, Leigh, Reichelt, Mike, Jones, Carol D., Zehnder, James L., Ito, Hideki, Arvin, Ann M.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: National Academy of Sciences 2007
Konular:
Biological Sciences
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC1955823/
https://ncbi.nlm.nih.gov/pubmed/17709745
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0706023104
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