Urocortin prevents mitochondrial permeability transition in response to reperfusion injury indirectly, by reducing oxidative stress

Urocortin (Ucn) protects hearts against ischemia and reperfusion injury whether given prior to ischemia or at reperfusion. Here we investigate the roles of protein kinase C, reactive oxygen species, and the mitochondrial permeability transition pore (MPTP) in mediating these effects. In Langendorff-...

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Detalhes bibliográficos
Main Authors: Townsend, Paul A., Davidson, Sean M., Clarke, Samantha J., Khaliulin, Igor, Carroll, Christopher J., Scarabelli, Tiziano M., Knight, Richard A., Stephanou, Anastasis, Latchman, David S., Halestrap, Andrew P.
Formato: Artigo
Idioma:Inglês
Publicado em: 2007
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1950441/
https://ncbi.nlm.nih.gov/pubmed/17483234
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.01135.2006
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