Absence of the β subunit (cchb1) of the skeletal muscle dihydropyridine receptor alters expression of the α(1) subunit and eliminates excitation-contraction coupling

Benzer Materyaller

• Recovery of Ca2+ current, charge movements, and Ca2+ transients in myotubes deficient in dihydropyridine receptor beta 1 subunit transfected with beta 1 cDNA.
  Yazar:: Beurg, M, ve diğerleri
  Baskı/Yayın Bilgisi: (1997)
• Molecular origin of the L-type Ca2+ current of skeletal muscle myotubes selectively deficient in dihydropyridine receptor beta1a subunit.
  Yazar:: Strube, C, ve diğerleri
  Baskı/Yayın Bilgisi: (1998)
• Reduced Ca2+ current, charge movement, and absence of Ca2+ transients in skeletal muscle deficient in dihydropyridine receptor beta 1 subunit.
  Yazar:: Strube, C, ve diğerleri
  Baskı/Yayın Bilgisi: (1996)
• Involvement of the carboxy-terminus region of the dihydropyridine receptor beta1a subunit in excitation-contraction coupling of skeletal muscle.
  Yazar:: Beurg, M, ve diğerleri
  Baskı/Yayın Bilgisi: (1999)
• Differential regulation of skeletal muscle L-type Ca2+ current and excitation-contraction coupling by the dihydropyridine receptor beta subunit.
  Yazar:: Beurg, M, ve diğerleri
  Baskı/Yayın Bilgisi: (1999)