The rheumatoid arthritis shared epitope increases cellular susceptibility to oxidative stress by antagonizing an adenosine-mediated anti-oxidative pathway

We have recently demonstrated that the rheumatoid arthritis (RA) shared epitope (SE) acts as a ligand that triggers nitric oxide (NO) signaling in opposite cells. Given the known pro-oxidative effect of NO and the proposed role of oxidative stress in the pathogenesis of RA, this study explores wheth...

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Kaydedildi:
Detaylı Bibliyografya
Asıl Yazarlar: Ling, Song, Li, Zhanguo, Borschukova, Olga, Xiao, Liqun, Pumpens, Paul, Holoshitz, Joseph
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: BioMed Central 2007
Konular:
Research Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC1865041/
https://ncbi.nlm.nih.gov/pubmed/17254342
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/ar2111
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