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Dosage-dependent switch from G protein-coupled to G protein-independent signaling by a GPCR
G-protein-coupled receptors (GPCRs) mostly signal through heterotrimeric G proteins. Increasing evidence suggests that GPCRs could function in a G-protein-independent manner. Here, we show that at low concentrations of an agonist, β(2)-adrenergic receptors (β(2)-ARs) signal through Gα(s) to activate...
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| Main Authors: | , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group
2007
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1782364/ https://ncbi.nlm.nih.gov/pubmed/17170700 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.emboj.7601502 |
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