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GLUT4 is internalized by a cholesterol-dependent nystatin-sensitive mechanism inhibited by insulin

Insulin slows GLUT4 internalization by an unknown mechanism. Here we show that in unstimulated adipocytes, GLUT4 is internalized by two mechanisms. Approximately 80% of GLUT4 is internalized by a mechanism that is sensitive to the cholesterol-aggregating drug nystatin, and is independent of AP-2 cla...

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Détails bibliographiques
Auteurs principaux: Blot, Vincent, McGraw, Timothy E
Format: Artigo
Langue:Inglês
Publié: 2006
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC1698906/
https://ncbi.nlm.nih.gov/pubmed/17139247
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.emboj.7601462
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