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Oncogenic potential of TASK3 (Kcnk9) depends on K(+) channel function
TASK3 gene (Kcnk9) is amplified and overexpressed in several types of human carcinomas. In this report, we demonstrate that a point mutation (G95E) within the consensus K(+) filter of TASK3 not only abolished TASK3 potassium channel activity but also abrogated its oncogenic functions, including prol...
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| Hoofdauteurs: | , , , , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
National Academy of Sciences
2003
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC164668/ https://ncbi.nlm.nih.gov/pubmed/12782791 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1232448100 |
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