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Saturated fatty acids inhibit induction of insulin gene transcription by JNK-mediated phosphorylation of insulin-receptor substrates

JNKs are attractive targets for treatment of obesity and type-2 diabetes. A sustained increase in JNK activity was observed in dietary and genetic models of obesity in mice, whereas JNK deficiency prevented obesity-induced insulin resistance. A similar insulin-sensitizing effect was seen upon treatm...

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Autori principali: Solinas, Giovanni, Naugler, Willscott, Galimi, Francesco, Lee, Myung-Shik, Karin, Michael
Natura: Artigo
Lingua:Inglês
Pubblicazione: National Academy of Sciences 2006
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC1637603/
https://ncbi.nlm.nih.gov/pubmed/17050683
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0607626103
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