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Functional in vivo interactions between JNK1 and JNK2 isoforms in obesity and insulin resistance
The c-Jun N-terminal kinases (JNKs) are key regulators of inflammation and interfere with insulin action in cultured cells and whole animals. Obesity increases total JNK activity, and JNK1, but not JNK2, deficiency results in reduced adiposity and improved insulin sensitivity. Interestingly, a highe...
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| Hlavní autoři: | , , , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
National Academy of Sciences
2006
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1487172/ https://ncbi.nlm.nih.gov/pubmed/16818881 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0603509103 |
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