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PLCγ2 regulates osteoclastogenesis via its interaction with ITAM proteins and GAB2
Excessive bone loss in arthritic diseases is mostly due to abnormal activation of the immune system leading to stimulation of osteoclasts. While phospholipase Cγ (PLCγ) isoforms are known modulators of T and B lymphocyte–mediated immune responses, we found that blockade of PLCγ enzymatic activity al...
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| Autors principals: | , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Society for Clinical Investigation
2006
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1616195/ https://ncbi.nlm.nih.gov/pubmed/17053833 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI28775 |
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