Intracellular Accumulation of Amyloidogenic Fragments of Amyloid-β Precursor Protein in Neurons with Niemann-Pick Type C Defects Is Associated with Endosomal Abnormalities

Схожие документы

• Loss of amyloid precursor protein exacerbates early inflammation in Niemann-Pick disease type C
  по: Shin, Samuel D., et al.
  Опубликовано: (2019)
• Accumulation of cholera toxin and GM1 ganglioside in the early endosome of Niemann–Pick C1-deficient cells
  по: Sugimoto, Yuko, et al.
  Опубликовано: (2001)
• ARF6-Mediated Endosome Recycling Reverses Lipid Accumulation Defects in Niemann-Pick Type C Disease
  по: Schweitzer, Jill Kuglin, et al.
  Опубликовано: (2009)
• Pathophysiological Roles of Amyloidogenic Carboxy-Terminal Fragments of the β-Amyloid Precursor Protein in Alzheimer’s Disease
  по: Keun-A Chang, et al.
  Опубликовано: (2005-01-01)
• Pathophysiological Roles of Amyloidogenic Carboxy-Terminal Fragments of the β-Amyloid Precursor Protein in Alzheimer’s Disease
  по: Keun-A Chang, et al.
  Опубликовано: (2005-01-01)