Caspase-Dependent Cleavage of c-Abl Contributes to Apoptosis
The nonreceptor tyrosine kinase c-Abl may contribute to the regulation of apoptosis. c-Abl activity is induced in the nucleus upon DNA damage, and its activation is required for execution of the apoptotic program. Recently, activation of nuclear c-Abl during death receptor-induced apoptosis has been...
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| Gepubliceerd in: | Mol Cell Biol |
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| Hoofdauteurs: | , , , , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Taylor & Francis
2003
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC152541/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12665579/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.23.8.2790-2799.2003 |
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