53BP1 and p53 synergize to suppress genomic instability and lymphomagenesis

p53-binding protein 1 (53BP1) participates in the cellular response to DNA double-stranded breaks where it associates with various DNA repair/cell cycle factors including the H2AX histone variant. Mice deficient for 53BP1 (53BP1(−/−)) are sensitive to ionizing radiation and immunodeficient because o...

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Main Authors: Morales, Julio C., Franco, Sonia, Murphy, Michael M., Bassing, Craig H., Mills, Kevin D., Adams, Melissa M., Walsh, Nicole C., Manis, John P., Rassidakis, George Z., Alt, Frederick W., Carpenter, Phillip B.
Formáid: Artigo
Teanga:Inglês
Foilsithe: National Academy of Sciences 2006
Ábhair:
Biological Sciences
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC1413919/
https://ncbi.nlm.nih.gov/pubmed/16492765
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0511259103
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