A C-Terminal Inhibitory Domain Controls the Activity of p63 by an Intramolecular Mechanism

The human genome is far smaller than originally estimated, and one explanation is that alternative splicing creates greater proteomic complexity than a simple count of open reading frames would suggest. The p53 homologue p63, for example, is a tetrameric transcription factor implicated in epithelial...

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Detaylı Bibliyografya
Asıl Yazarlar: Serber, Zach, Lai, Helen C., Yang, Annie, Ou, Horng D., Sigal, Martina S., Kelly, Alexander E., Darimont, Beatrice D., Duijf, Pascal H. G., van Bokhoven, Hans, McKeon, Frank, Dötsch, Volker
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Society for Microbiology 2002
Konular:
Transcriptional Regulation
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC139862/
https://ncbi.nlm.nih.gov/pubmed/12446779
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.22.24.8601-8611.2002
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