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A C-Terminal Inhibitory Domain Controls the Activity of p63 by an Intramolecular Mechanism

The human genome is far smaller than originally estimated, and one explanation is that alternative splicing creates greater proteomic complexity than a simple count of open reading frames would suggest. The p53 homologue p63, for example, is a tetrameric transcription factor implicated in epithelial...

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Publicat a:Mol Cell Biol
Autors principals: Serber, Zach, Lai, Helen C., Yang, Annie, Ou, Horng D., Sigal, Martina S., Kelly, Alexander E., Darimont, Beatrice D., Duijf, Pascal H. G., van Bokhoven, Hans, McKeon, Frank, Dötsch, Volker
Format: Artigo
Idioma:Inglês
Publicat: Taylor & Francis 2002
Matèries:
Transcriptional Regulation
Accés en línia:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC139862/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12446779/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.22.24.8601-8611.2002
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