A C-Terminal Inhibitory Domain Controls the Activity of p63 by an Intramolecular Mechanism

The human genome is far smaller than originally estimated, and one explanation is that alternative splicing creates greater proteomic complexity than a simple count of open reading frames would suggest. The p53 homologue p63, for example, is a tetrameric transcription factor implicated in epithelial...

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書誌詳細

出版年:	Mol Cell Biol
主要な著者:	Serber, Zach, Lai, Helen C., Yang, Annie, Ou, Horng D., Sigal, Martina S., Kelly, Alexander E., Darimont, Beatrice D., Duijf, Pascal H. G., van Bokhoven, Hans, McKeon, Frank, Dötsch, Volker
フォーマット:	Artigo
言語:	Inglês
出版事項:	Taylor & Francis 2002
主題:	Transcriptional Regulation
オンライン･アクセス:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC139862/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12446779/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.22.24.8601-8611.2002
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