A C-Terminal Inhibitory Domain Controls the Activity of p63 by an Intramolecular Mechanism

The human genome is far smaller than originally estimated, and one explanation is that alternative splicing creates greater proteomic complexity than a simple count of open reading frames would suggest. The p53 homologue p63, for example, is a tetrameric transcription factor implicated in epithelial...

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Détails bibliographiques
Auteurs principaux: Serber, Zach, Lai, Helen C., Yang, Annie, Ou, Horng D., Sigal, Martina S., Kelly, Alexander E., Darimont, Beatrice D., Duijf, Pascal H. G., van Bokhoven, Hans, McKeon, Frank, Dötsch, Volker
Format: Artigo
Langue:Inglês
Publié: American Society for Microbiology 2002
Sujets:
Transcriptional Regulation
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC139862/
https://ncbi.nlm.nih.gov/pubmed/12446779
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.22.24.8601-8611.2002
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