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A C-Terminal Inhibitory Domain Controls the Activity of p63 by an Intramolecular Mechanism
The human genome is far smaller than originally estimated, and one explanation is that alternative splicing creates greater proteomic complexity than a simple count of open reading frames would suggest. The p53 homologue p63, for example, is a tetrameric transcription factor implicated in epithelial...
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| Päätekijät: | , , , , , , , , , , |
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| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
American Society for Microbiology
2002
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC139862/ https://ncbi.nlm.nih.gov/pubmed/12446779 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.22.24.8601-8611.2002 |
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