A C-Terminal Inhibitory Domain Controls the Activity of p63 by an Intramolecular Mechanism

The human genome is far smaller than originally estimated, and one explanation is that alternative splicing creates greater proteomic complexity than a simple count of open reading frames would suggest. The p53 homologue p63, for example, is a tetrameric transcription factor implicated in epithelial...

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Detalles Bibliográficos

Publicado en:	Mol Cell Biol
Principais autores:	Serber, Zach, Lai, Helen C., Yang, Annie, Ou, Horng D., Sigal, Martina S., Kelly, Alexander E., Darimont, Beatrice D., Duijf, Pascal H. G., van Bokhoven, Hans, McKeon, Frank, Dötsch, Volker
Formato:	Artigo
Idioma:	Inglês
Publicado:	Taylor & Francis 2002
Assuntos:	Transcriptional Regulation
Acceso en liña:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC139862/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12446779/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.22.24.8601-8611.2002
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