Impaired Cardiac Contractility Response to Hemodynamic Stress in S100A1-Deficient Mice
Ca(2+) signaling plays a central role in cardiac contractility and adaptation to increased hemodynamic demand. We have generated mice with a targeted deletion of the S100A1 gene coding for the major cardiac isoform of the large multigenic S100 family of EF hand Ca(2+)-binding proteins. S100A1(−/−) m...
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| 發表在: | Mol Cell Biol |
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| Principais autores: | , , , , , , |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
Taylor & Francis
2002
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC133731/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11909974/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.22.8.2821-2829.2002 |
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