The yeast counterparts of human 'MELAS' mutations cause mitochondrial dysfunction that can be rescued by overexpression of the mitochondrial translation factor EF-Tu

Lignende værker

• A Mutation in a Novel Yeast Proteasomal Gene, RPN11/MPR1, Produces a Cell Cycle Arrest, Overreplication of Nuclear and Mitochondrial DNA, and an Altered Mitochondrial Morphology
  af: Rinaldi, Teresa, et al.
  Udgivet: (1998)
• Yeast as a model of human mitochondrial tRNA base substitutions: Investigation of the molecular basis of respiratory defects
  af: Montanari, Arianna, et al.
  Udgivet: (2008)
• A point mutation in a mitochondrial tRNA gene abolishes its 3' end processing.
  af: Zennaro, E, et al.
  Udgivet: (1989)
• Novel mutation in mitochondrial Elongation Factor EF-Tu associated to dysplastic leukoencephalopathy and defective mitochondrial DNA translation
  af: Di Nottia, Michela, et al.
  Udgivet: (2017)
• Aminoacylation and conformational properties of yeast mitochondrial tRNA mutants with respiratory deficiency
  af: FRANCISCI, SILVIA, et al.
  Udgivet: (2005)