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Interaction of Avilamycin with Ribosomes and Resistance Caused by Mutations in 23S rRNA
The antibiotic growth promoter avilamycin inhibits protein synthesis by binding to bacterial ribosomes. Here the binding site is further characterized on Escherichia coli ribosomes. The drug interacts with domain V of 23S rRNA, giving a chemical footprint at nucleotides A2482 and A2534. Selection of...
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| Main Authors: | , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Microbiology
2002
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| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC128742/ https://ncbi.nlm.nih.gov/pubmed/12384333 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/AAC.46.11.3339-3342.2002 |
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