Wound-healing defect of CD18(−/−) mice due to a decrease in TGF-β(1) and myofibroblast differentiation

We studied the mechanisms underlying the severely impaired wound healing associated with human leukocyte-adhesion deficiency syndrome-1 (LAD1) using a murine disease model. In CD18(−/−) mice, healing of full-thickness wounds was severely delayed during granulation-tissue contraction, a phase where m...

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Bibliografische gegevens
Hoofdauteurs: Peters, Thorsten, Sindrilaru, Anca, Hinz, Boris, Hinrichs, Ralf, Menke, André, Al-Azzeh, Ezz Al Din, Holzwarth, Katrin, Oreshkova, Tsvetelina, Wang, Honglin, Kess, Daniel, Walzog, Barbara, Sulyok, Silke, Sunderkötter, Cord, Friedrich, Wilhelm, Wlaschek, Meinhard, Krieg, Thomas, Scharffetter-Kochanek, Karin
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: 2005
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Article
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC1276170/
https://ncbi.nlm.nih.gov/pubmed/16148944
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.emboj.7600809
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